N	Concordance
1	h low resting metabolic rate, abdominal obesity, increased lipid oxidation, hype
2	creasingly important predictor of adult obesity, regardless of whether the paren
3	 metabolism in patients with alimentary obesity during therapy with monosodium g
4	usceptible to epidermal hyperplasia and obesity in a mixed genetic background. W
5	 and men more likely to exhibit central obesity, carrying weight "within" their 
6	mographic factors and risk of childhood obesity provides important clues for und
7	 in prepubertal children with exogenous obesity at the time of clinical diagnosi
8	incidence of parental and grandparental obesity was determined from patient hist
9	al whether metabolic disorders of human obesity include protein metabolism. Even
10	 predisposition to develop diet-induced obesity (DIO). This study used quantitat
11	nges, are null mutations causing morbid obesity, because homozygotes for the var
12	critical periods for the development of obesity that persists into adulthood. No
13	 been associated with genetic models of obesity and insulin resistance. Because 
14	e data on time-trends the prevalence of obesity seems to be increasing. In most 
15	ts. In contrast, effective treatment of obesity results in a substantial decreas
16	 obesity in adulthood, but how parental obesity affects the chances of a child's
17	erences in obesity, specifically severe obesity. This was a population-based, re
18	insulinemia is commonly associated with obesity, but it has not been determined