N	Concordance
1	ground of the poor prognosis of stage 4 neuroblastoma and lack of other effectiv
3	ytogenetic and molecular changes. Adult neuroblastomas are rare, and their relat
4	ctively monitors children with advanced neuroblastoma, radiographically and with
5	  subcutaneous injections of autologous neuroblastoma cells that have been  modi
6	countered two cases of primary cervical neuroblastoma in infants. The first case
7	n described in rare cases of congenital neuroblastoma, usually in association wi
8	ction, various numbers of contaminating neuroblastoma cells were found in the le
9	f affected children have differentiated neuroblastomas, usually located in the t
10	osition to and pathogenesis of familial neuroblastoma and potentially sporadic t
11	ic expression of reporter gene in IMR32 neuroblastoma cells while remaining sile
13	ozygosity at chromosome 9p21 in primary neuroblastomas: evidence for two deleted
14	f treatment of recurrent or progressive neuroblastoma depends on  the location a
15	n children with refractory or recurrent neuroblastoma or  Wilms' tumor.  II.  As
16	rogression in  patients with refractory neuroblastoma treated with topotecan wit
17	    therapy for children with high-risk neuroblastoma: a phase I/II study of    
18	tcome for patients  with advanced-stage neuroblastoma when treated with myeloabl