N	Concordance
1	lly treated with chemotherapy for acute myeloid leukemia (AML), whereas three ot
2	  primary refractory or recurrent acute myeloid leukemia or secondary acute  mye
3	horus, only 1 developed secondary acute myeloid leukemia (AML) 9 years afterward
4	kemic cells from 21 patients with acute myeloid leukemia (AML), and 6 patients w
5	of patients treated for childhood acute myeloid leukemia comes an increased awa
6	lysis of minimally differentiated acute myeloid leukemia (AML, M0). We performed
7	quiescence associated antigens in acute myeloid leukemia correlates with surviva
8	bitors. The other two had de novo acute myeloid leukemia or myelodysplastic synd
9	rrow transplantation (BMT), adult acute myeloid leukemia (AML) has become a cura
10	 the myelodysplastic syndrome and acute myeloid leukemia following melphalan and
11	 Recently we observed a case of chronic myeloid leukemia (CML) with t(9;22) afte
12	hiladelphia chromosome positive chronic myeloid leukemia in chronic or accelera
13	ute lymphocytic leukemia (ALL), chronic myeloid leukemia (CML), and acute myeloi
14	 of patients with chronic phase chronic myeloid leukemia (CML) after treatment w
15	 which increased up to 180 min in human myeloid leukemia U937 cells. Inactive an
16	or in regulating the differentiation of myeloid leukemia cells. The results also