N	Concordance
1	ection, we stably transfected RAW 264.7 macrophages with a rat cyclooxygenase-2 
2	 novel anti-rheumatic drug TA-383 has a macrophage migration enhancing activity.
3	of 1,25-dihydroxyvitamin D by activated macrophages has been shown to be the cau
4	integrin-mediated adhesion. In adherent macrophages, absence of CD45 led to the 
5	vation. Normal human pulmonary alveolar macrophages (PAM) are less sensitive to 
6	 relationship between tumour associated macrophages (TAM) and apoptotic cells in
7	 production in primary peripheral blood macrophages and fetal brains (astrocytes
8	croglial lineages. Recruitment of brain macrophages is promoted by transformatio
9	n of CD11a on cultured monocyte-derived macrophages (MDMs). Monocytes isolated f
10	taining cellular debris, numerous foamy macrophages, and other inflammatory cell
11	of their differentiation by granulocyte macrophage-colony stimulating factor and
12	collected. The MCP-1 mRNA expression in macrophages was examined by Northern blo
13	atin and pravastatin, on Ox-LDL-induced macrophage growth. Our results demonstra
14	mination of the infection. HIV-infected macrophages have been demonstrated not o
15	expression was elicited in inflammatory macrophages and in distinct cardiomyocyt
16	n EGF-like growth factor by parenchymal macrophages following bleomycin-induced 
17	. The cytostatic activity of peritoneal macrophages was higher in mice treated w
18	h LPS. Cell lysates from LPS-stimulated macrophages exhibited a markedly increas