N Concordance 1 ome, eosinophilic polymyositis and Ki-1 lymphoma on pathogenetic mechanism. 2 teristics of the core filaments in EL-4 lymphoma cells may reflect cell-type spe 3 an comparably-staged B-cell aggressive lymphomas [43,44], while other investiga 4 of Ki-1-positive large-cell anaplastic lymphoma in an 87-year-old man diagnosed 5 noblastic T-cell lymphoma, angiocentric lymphoma, intestinal T-cell lymphoma, p 6 l anorectal location of AIDS-associated lymphomas is explainable by the high inc 7 effusion lymphomas or body cavity-based lymphomas (BCBLs). A latency-associated 8 motherapy, patients with primary brain lymphoma receive a course of cranial irr 9 B cells and the EBV-positive Burkitt's lymphoma cell line, Raji, as activators 10 e. We present a case of primary cardiac lymphoma initially diagnosed antemortem 11 , mostly non-Hodgkin's low grade B cell lymphomas. In this report a case is desc 12 phoma is a subset of diffuse large cell lymphoma characterized by significant f 13 ar evidence of apoptosis in mantle cell lymphoma cells from patients before and 14 erent from that of the cutaneous T-cell lymphomas and Sezary syndrome, even in T 15 and follicular centroblast-centrocytic lymphoma were not found. Activity of ami 16 n small- and large-cell primary gastric lymphoma using a specific staining metho 17 7 patients with low-grade non-Hodgkin's lymphoma (NHL) and to 19 patients with C 18 gnosing mediastinal spread of malignant lymphomas and has interesting potentials 19 ry and response to therapy. The nodular lymphomas generally behaved in an indole