N	Concordance
1	on between the intrinsic heme a/heme a3 electron-transfer rate and equilibrium c
2	nction. Both NADH and NADPH were active electron donors in this system, while NA
3	lized areas of relapse with  additional electron-beam irradiation or possibly to
4	r and non-lung cancer cases. Analytical electron microscopic studies identified 
5	inone and decylubiquinone as artificial electron acceptors. We have found that t
6	th the material-dependent backscattered electron signal that shows the platinum 
7	nal map of frog rhodopsin determined by electron cryo-microscopy. The model sugg
8	ts quantification by gas chromatography-electron capture chemical ionization mas
9	cles were detected by negative contrast electron microscopy of extracts from the
10	ry angiography and by contrast enhanced electron beam CT scan. Simple statistic 
11	 was involved. Although freeze-fracture electron microscopy of stressed and unst
12	a to Calpha. This is driven by the high electron density of residues (Glu199 and
13	ntly. Immunofluorescence and immunogold electron microscopy showed that followin
14	djacent  structures.[1]  Intraoperative electron beam radiotherapy, to the site 
15	death due to treatment of mitochondrial electron transport inhibitors including 
16	ned to undergo a hypoxia-dependent, one-electron reduction to metabolites that a
17	e-resolved EPR in studying photoinduced electron and energy transfer in isotropi
18	eld microscopy, field emission scanning electron microscopy, and optical density
19	M) equipped with the standard secondary electron (SE) detector was used to image
20	of the skin.  This is called total skin electron  beam radiation therapy, or TSE
21	educed ability of H193A to catalyze two-electron hydroperoxide reduction resulte