N	Concordance
1	 PMNs was not detected until 72 h after dexamethasone injection. These data prov
2	istration of the glucocorticoid agonist dexamethasone (DEX) using osmotic minipu
3	 Natural and synthetic hormones such as dexamethasone, aldosterone, and thyroxin
4	mens. High-dose glucocorticoids such as dexamethasone are often used for brief i
5	II mRNA levels in PBMCs and PMNs before dexamethasone treatment. This is in cont
6	erived cell line HaCaT was inhibited by dexamethasone, suggesting that the anti-
7	atients with this syndrome is high dose dexamethasone at 10mg every 12 hours for
8	ese cells, the synthetic glucocorticoid dexamethasone exerts a dominant repressi
9	urse single-dose regimen of intravenous dexamethasone, diphenhydramine, and cime
10	 was observed between administration of dexamethasone and MGP inhibition. This i
11	gic inflammation to study the effect of dexamethasone on eosinophil recruitment.
12	ns and administration of high-dose oral dexamethasone was followed by a marked a
13	ed with longer durations of exposure to dexamethasone and with higher concentrat
14	 to basal levels, and pretreatment with dexamethasone completely abolished this