N	Concordance
1	+ channel blockers in models of central nervous system disease in animals and in
2	f these substances to the human central nervous system (CNS) remains problematic
3	ll damage and cell death in the central nervous system in particular. To determi
4	on-Burkitt subtypes) or primary central nervous system lymphoma.[1]    AIDS-rel
5	abundant glial cell type of the central nervous system that appear to play a rol
6	rbidity burden of survival from central nervous system (CNS) tumours and its imp
7	tions of various regions of the central nervous system of neonatal and adult cat
8	Histologically proven high-risk central nervous system embryonal tumors such  as
9	l of these steroids in that the central nervous system (CNS) also possesses enzy
10	ntly expressed in the postnatal central nervous system (CNS) but barely detectab
11	on in children can cause severe central nervous system depression and cardiovasc
12	 ignant germ cell tumors of the central nervous system in order to provide sampl
13	ious studies that the marsupial central nervous system is born at a relatively i
14	e affinity of the agent for the central nervous system is controlled. Here we sh