N	Concordance
1	05). In conclusion, serum bone alkaline phosphatase should be preferred to bone 
2	tein kinase A and inhibited by alkaline phosphatase treatment at all ages, prote
3	ted using the Escherichia coli alkaline phosphatase signal sequence.            
4	brane attachment of endogenous alkaline phosphatase. Similarly, in cells cotrans
5	r peroxidase or dioxethane for alkaline phosphatase. The kinase assay was carrie
6	und including amylase, lipase, alkaline phosphatase, total bilirubin, GOT, and G
7	ytic enzyme tissue-nonspecific alkaline phosphatase (TN-AP) and the placental al
8	e phosphatase: The activity of alkaline phosphatase was also significantly affec
9	Y, and defective maturation of alkaline phosphatase. Subcellular fractionation a
10	t as a substitute activator of alkaline phosphatase, the degree of compensation 
11	used with the secreted form of alkaline phosphatase (SLC-SEAP) was used to chara
12	 bryonic antigen and placental alkaline phosphatase. The immunohistoc-       hem
13	. A mild increase in the serum alkaline phosphatase value is the most common fin
14	compared serum levels of total alkaline phosphatase (TAP) and bone-specific alka